Cubism -the Weeping Woman Essay Example for Free

Cubism -the Weeping Woman Essay Cubism was an art movement which originated in France and Spain in 1906. Cubism influenced painting movement. Cubist artists include Pablo Picasso, George Braque and Juan Gris. Picasso had recently travelled to Africa and native America and was inspired by the tribal masks. Cubist Artists captured different view points at the same time. This showed collage and made the image look 3D. In the painting ‘The Weeping Woman’ she looks like she is at home. I get this idea from the border behind her in the middle of what looks like two walls. I also think it looks homely because it is painted yellow, a colour associated with warmth and comfort. It could also be a church, the brown strip could symbolise the wooden seats in a church. Another thing it could symbolise, is the attendance of a funeral because she is wearing black coloured clothing. In the middle of her face, which is the colour white/blue, it could mean a part of her being taken away, not only is her colour draining away, but so are her emotions. She seems to be hiding her sadness. You can tell she is sad and upset because her eyes look glazed over and there is a red/pink blush under her eyes and nose which could show that she has been crying and has wiped her tears away. The sharp jagged lines could represent how she is holding herself together, and maybe the other emotions she feels, like anger. You can see that she is in distress as she has creases on her forehead. The bits of green blended in with the yellow could represent jealousy, but could also symbolise being ill or sick. She is so upset that its actually making her feel weak and unwell. The white could also be seen as being some kind of ghostly figure, which links back to the thought of her attending a funeral, and why she is so upset. The green representing jealousy, could be envy for other people who’s loved ones are still with them. Her hands look like they are together, like she is trying to pray, for the protection of her loved one in the afterlife. She could also be holding something, like a tissue to wipe away her tears, a possession of the bereaved, a letter or a will. This painting was actually painted representing the weeping woman as a mother of a soldier who had died at war. This connects to the thing she could be holding, as when a soldier dies at war, they usually send a letter to inform the close family of this.

Cerebral Autoregulation Mechanism | Report

Cerebral Autoregulation Mechanism | Report From: Biose Ifechukwude Joachim Introduction Cerebral autoregulation (CA) is the multifactorial vascular mechanism that maintains a constant cerebral blood supply in spite of fluctuations in the cerebral perfusion pressure (CPP) (Lassen, 1959; Tiecks et al., 1995). This mechanism thrives for CPP values within the range of 50-150 mmHg (Lassen, 1959; Paulson, Strandgaard and Edvinsson, 1990; Panerai, 1998) (Fig. 1). The vascular response involved in CA is rapid and so robust that hypertension (Eames et al., 2003; Serrador et al., 2005; Zhang et al., 2007) and aging (Eames et al., 2003; Fisher et al., 2008; Liu et al., 2013; Oudegeest-Sander et al., 2014) does not alter its physiological role. However, CA is compromised following pathologic conditions such as traumatic brain injury, intracerebral haemorrhage, stroke, hyper-perfusion syndrome, and subarachnoid haemorrhage (Diedler et al., 2009; Atkins et al., 2010; Budohoski et al., 2012; Saeed et al., 2013; Buczek et al., 2013). Fig. 1. Cerebral autoreglation in relation to vascular response. Within the upper and lower boundaries of the autoregulatory range (dotted lines), blood flow remains constant (blue line with beads). As Pressure falls below the lower limit, vascular smooth muscle relaxes to allow dilatation, while constriction of vessels (red circles) ensues to reduce blood flow as pressure approximates the upper limit. Adapted from Pires et al., 2013. Classification Based on factors affecting cerebral blood flow (CBF), CA can be classified into two categories, metabolic autoregulation (MA) and pressure autoregulation (PA). Mainly due to changes in brain tissue pH (Cotev and Severinghaus, 1969; Betz and Heuser, 1967; Raichle, Posner and Plum, 1970), MA is the principal regulatory mechanism of CBF according to metabolic demand. This implies that MA responds to local or global ischemia and hypoxia which increases pH by increasing CBF via vasodilatation (Ekstrom-Jodal et al., 1971; Raichle and Stone, 1971).While PA is the vascular response to maintain blood flow following changes in perfusion pressure, achieved by varying the degree of vasoconstriction or vasodilatation of the cerebral vasculature. Mechanism In adults and under normal conditions, provided CPP falls within the boundary of 50-150 mmHg, CBF is preserved at approximately 50 mL per 100 g of brain tissue per minute (McHenry et al., 1974; Strandgaard et al., 1976; Paulson, Strandgaard and Edvinsson, 1990). Outside this range of CPP, CA is impaired and CBF becomes directly dependent on mean arterial pressure (MacKenzie et al., 1976; Heistad and Kontos, 1979; Baumbach and Heistad, 1985; Paulson et al., 1990). More so, should CPP falls below the lower boundary of CA, blood flow reduces and ischemia sets in (Hossmann, 2006). The precise mechanism of CA is currently elusive; however, it is believed to be subject to the interaction of neurogenic, metabolic and myogenic factors (Czosnyka et al., 2009; Novak and Hajjar, 2010). Intrinsic innervation is touted to be directly involved in the mechanisms of CA (Goadsby and Edvinsson, 2002) and extrinsic pathway is implausible, since CA is unimpaired following sympathetic and parasympathetic denervation in experimental animals (Busija and Heistad, 1984). The perikarya within the subcortical region of the brain, precisely those from the nucleus basalis, locus ceruleus and raphe nucleus project to cortical microvessels for the control of local blood flow by release of neurotransmitters (ACH, norepinephrine and 5HT) (Hamel, 2006). These released neurotransmitter substances interact with the receptors on smooth muscle, endothelium, or astrocytes to cause constriction or dilation, thus regulating blood supply according to the metabolic demand (Iadecola, 2004; Hamel, 2006; Drake and Iadecola, 2007). Also, metabolic by-products released by the brain during CBF decrease are important for CA (Paulson, Strandgaar and Edvinsson, 1990). These substances, potassium, adenosine, and hydrogen ion triggers vasodilatation. Another important component of the CA mechanism is the myogenic response of the cerebrovascular smooth muscle in regulating vascular tone. Constriction of the cerebral vasculature due to smooth muscle contraction ensues during pressure fluctuations at the upper boundary of the autoregulatory range of CPP, thus blood flow is not excessive (Fig. 1). Conversely, fluctuations at the lower limit of CPP is followed by vasodilatation (Fig.1) (Kontos, 1978,Busija and Heistad, 1984; Mellander, 1989; Osol et al., 2002). Furthermore, the direct contact between astrocytes and the parenchymal arterioles of the brain have been shown to play a role in CA (Rennels and Nelson, 1975; Cohen, Molinatti and Hamel, 1997; Iadecola, 2004; Hamel, 2006; Drake and Iadecola, 2007; Zlokovic, 2008). Most microvessels at the subcortical level have astrocytic end-feet at the interface between them and neurons (Kulik et al., 2008), thus, under the direct influence of the vasoactive factors released by astrocytes (Murphy et al., 1994). Interestingly, the type of cerebral vasculature may also contribute to CA in an unexpected manner, with respect to their response to blood flow changes. While basilar artery dilates in response to increased blood flow, MCA constricts Koller and Toth, (2012). Under Anaesthesia Anaesthesia puts the brain in a state of reduced neuronal activity, as a result CBF decreases in light of neurovascular coupling (Attwell et al., 2010). Also, in their studies in rats, Jones et al., (2002) reported that anaesthesia reduces the CCP levels below the lower limit of CA. More importantly, anaesthetics have significant impact on CA as they affect the vasculature of the brain, directly or indirectly. Under the influence of volatile anaesthetics, calcium entry via voltage gated Ca2+ channels on vascular smooth muscle cells is reduced significantly, causing the vasculature to dilate (Bosnjak et al. 1992), thereby, directly overriding CA. Also, anaesthetics cause profound respiratory depression in spontaneously breathing animals, consequently PaCO2 increased. Given that the vasculature of the brain is highly sensitive to changes in CO2, an increase value of PaCO2 stimulates cerebral vasodilatation (Kuschinsky, 1997; Willie et al., 2014); correspondingly CBF increases (Figure 2). These effects of anaesthetics lead ultimately to the failure of CA in mammals. However, certain anaesthetics for example Ethomidate, preserves CA (Wang et al., 2010). This is mainly due to their ability to keep PaCO2 nearly constant within the nomal range without artificial ventilation (Lacombe et al. 2005; Joutel et al., 2010). Fig. 2. Cerebral blood flow with respect to arterial pressure of CO2. CBF increases as PaCO2 level increases beyond the level of 25 mmHg. However, at 80 mmHg blood vessels are maximally dilated and CBF remains constant with a further increase in PaCO2 values. Adapted from Adapted from Hill and Gwinnutt, no date. Stroke During arterial occlusion, as in the case of ischaemic stroke, local cerebral perfusion pressure falls below the normal CA range while MAP does not change. With persistent occlusion, autoregulation fails (Reinhard et al., 2008; Reinhard et al., 2012; Immink et al., 2005; Atkins et al., 2010) and regional CBF further decreases. For this reason, blood pressure changes, high or low, results in poor outcome (Castillo et al, 2004; Aslanyan et al., 2003; Sandset et al., 2012). However, this is not entirely due to the failed autoregulatory capacity of the vessels during ischemia, but perhaps their normal vasodilatory capacity has reached a maximal limit (Petersen et al., 2015). The impaired autoregulatory response following acute stroke has been observed both in the affected and contralateral hemispheres (Cupini et al., 2001; Dawson et al., 2000; Dawson, Panerai and Potter, 2003; Fieschi et al., 1988; Gelmers, 1982; Lisk et al., 1993; Hakim et al., 1989). 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Differential effects of halothane, enflurane, and isofluurane on Ca2 + transients and papillary muscle tension in guinea pigs. Anesthesiology. 76: 123–131 Buczek J, Karlin ´ski M, Kobayashi A, BiaÅ‚ek P and CzÅ‚onkowska A (2013). Hyperperfusion syndrome after carotid endarterectomy and carotid stenting. Cerebrovasc. Dis. 35: 531–7. Budohoski KP, Czosnyka M, Smielewski P, Kasprowicz M, Helmy A, Bulters D et al. (2012). Impairment of cerebral autoregulation predicts delayed cerebral ischemia after subarachnoid hemorrhage: a prospective observational study. Stroke. 43: 3230–3237. Busija DW and Heistad DD (1984). Factors involved in the physiological regulation of the cerebral circulation. Rev. Physiol. Biochem. Parmacol. 101: 161–211. Castillo J, Leira R, Garcà ­a MM, Serena J, Blanco M and Dà ¡valos A (2004). Blood pressure decrease during the acute phase of ischemic stroke is associated with brain injury and poor stroke outcome. Stroke. 35: 520–526. Cohen Z, Molinatti G and Hamel E (1997). Astroglial and vascular interactions of noradrenaline terminals in the rat cerebral cortex. J. Cereb. Blood Flow Metab. 17: 894–904. Cotev S and Severinghaus JW (1969). Role of cerebrospinal fluid pH in management of respiratory problems. Anesth. Analg. 48: 42-47. Cupini LM, Diomedi M, Placidi F, Silvestrini M and Giacomini P (2001). Cerebrovascular reactivity and subcortical infarctions. Arch. Neurol. 58: 577–581. Czosnyka M, Brady K, Reinhard M, Smielewski P and Steiner LA (2009). Monitoring of cerebrovascular autoregulation: facts, myths, and missing links. Neurocritical Care. 10: 373–86. Dawson SL, Blake MJ, Panerai RB and Potter JF (2000). Dynamic but not static cerebral autoregulation is impaired in acute ischaemic stroke. Cerebrovasc. Dis.10:126–132. Dawson SL, Panerai RB and Potter JF (2003). Serial changes in static and dynamic cerebral autoregulation after acute ischaemic stroke. Cerebrovasc. Dis. 16:69–75. 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Immink RV, van Montfrans GA, Stam J, Karemaker JM, Diamant M and van Lieshout JJ (2005). Dynamic cerebral autoregulation in acute lacunar and middle cerebral artery territory ischemic stroke. Stroke. 36: 2595–2600. Jones SC, Radinsky CR, Furlan AJ et al. (2002). Variability in the magnitude of the cerebral blood flow response and the shape of the cerebral blood flow pressure autoregulation curve during hypotension in normal rats [corrected]. Anesthesiology. 97: 488–96. Joutel A, Monet-Lepretre M, Gosele C, Baron-Menguy C, Hammes A, Schmidt S, Lemaire-Carrette B, Domenga V, Schedl A, Lacombe P and Hubner N (2010). Cerebrovascular dysfunction and microcirculation rarefaction precede white matter lesions in a mouse genetic model of cerebral ischemic small vessel disease. J. Clin. Invest. 120: 433–445. Koller A and Toth P (2012). Contribution of flow-dependent vasomotor mechanisms to the autoregulation of cerebral blood flow. J. Vasc. Res. 49: 375–389. Kontos HA, Wei EP, Navari RM, Levasseur JE, Rosenblum WI and Patterson JL, Jr (1978). Responses of cerebral arteries and arterioles to acute hypotension and hypertension. Am. J. Physiol. 234: H371–H383. Kulik T, Kusano Y, Aronhime S, Sandler AL and Winn HR (2008). Regulation of cerebral vasculature in normal and ischemic brain. Neuropharmacology. 55: 281–288. Kuschinsky W (1997). Neuronal-vascular coupling. A unifying hypothesis. Adv. Exp. Med. Biol. 413: 167–176. Lacombe P, Oligo C, Domenga V, Tournier-Lasserve E and Joutel A (2005). Impaired cerebral vasoreactivity in a transgenic mouse model of cerebral autosomal dominant arteriopathy with subcortical infarcts and leukoencephalopathy arteriopathy. Stroke. 36: 1053–1058. Lassen NA (1959).Cerebral blood flow and oxygen consumption in man. Physiol. Rev. 39: 183–238. Lassen NA (1974). Control of cerebral circulation in health and disease. Circ. Res. 34: 749–760. Lisk DR, Grotta JC, Lamki LM, Tran HD, Taylor JW, Molony DA and Barron BJ (1993). Should hypertension be treated after acute stroke? A randomized controlled trial using single photon emission computed tomography. Arch. Neurol. 50:855–862. Liu J, Zhu YS, Hill C, Armstrong K, Tarumi T, Hodics T, Hynan LS and Zhang R (2013). Cerebral autoregulation of blood velocity and volumetric flow during steady-state changes in arterial pressure. Hypertension 62: 973– 979. MacKenzie ET, Strandgaard S and Graham DI et al. (1976). Effects of acutely induced hypertension in cats on pial arteriolar caliber, local cerebral blood flow, and the blood-brain barrier. Circ. Res. 39:33-41. McHenry LC, Jr., West JW, Cooper ES, Goldberg HI and Jaffe ME (1974).Cerebral autoregulation in man. Stroke. 5: 695-706. Mellander S (1989). Functional aspects of myogenic vascular control. J. Hypertens. 7(4): S21–S30. Murphy S, Rich G, Orgren KI, Moore SA and Faraci FM (1994). Astrocyte-derived lipoxygenase product evokes endothelium-dependent relaxation of the basilar artery. J. Neurosci. Res. 38: 314–318. Novak V and Hajjar I (2010). The relationship between blood pressure and cognitive function. Nature Reviews Cardiology. 7: 686–98. Osol G, Brekke JF, McElroy-Yaggy K and Gokina NI (2002). Myogenic tone, reactivity, and forced dilatation: a three-phase model of in vitro arterial myogenic behavior. Am. J. Physiol. Heart Circ. Physiol. 283: H2260– H2267. Oudegeest-Sander MH, van Beek AH, Abbink K, Olde Rikkert MG, Hopman MT and Claassen JA (2014). Assessment of dynamic cerebral autoregulation and cerebrovascular CO2 reactivity in ageing by measurements of cerebral blood flow and cortical oxygenation. Exp Physiol. 99: 586–598. Panerai RB (1998). Assessment of cerebral pressure autoregulation in humans—a review of measurement methods. Physiol. Meas. 19: 305–338. Paulson OB, Strandgaard S and Edvinsson L (1990). Cerebral autoregulation. Cerebrovasc. Brain Metab. Rev. 2: 161-192. Petersen NH, Ortega-Gutierrez S, Reccius A, Masurkar A, Huang A and Marshall RS (2015). Dynamic Cerebral Autoregulation Is Transiently Impaired for One Week after Large-Vessel Acute Ischemic Stroke. Cerebrovasc. Dis. 39: 144–150. Pires PW, Dams Ramos CM, Matin N and Dorrance AM (2013). The effects of hypertension on the cerebral circulation. Am. J. Physiol. Heart. Circ. Physiol. 304: 1598–1614, Raichle ME and Stone HL (1971). Cerebral blood flow autoregulation and graded hypercapnia. Eur. Neurol. 6: 1-5. Reinhard M, Wihler C, Roth M, Harloff A, Niesen WD, Timmer J et al. (2008). Cerebral autoregulation dynamics in acute ischemic stroke after rtPA thrombolysis. Cerebrovasc. Dis. 26: 147–155. Reinhard M, Rutsch S, Lambeck J, Wihler C, Czosnyka M, Weiller C et al. (2012). Dynamic cerebral autoregulation associates with infarct size and outcome after ischemic stroke. Acta  Neurol. Scand.125: 156–162.   Rennels M and Nelson E (1975). Capillary innervation in the mammalian central nervous system: an electron microscope demonstration (1). Am. J. Anat. 144: 233–241. Saeed NP, Panerai RB and Robinson TG (2013). The carotid artery as an alternative site to the middle cerebral artery for reproducible estimates of autoregulation index. Ultrasound Med. Biol. 39: 735–741. Sandset EC, Murray GD, Bath PM, Kjeldsen SE and Berge E (2012). Scandinavian Candesartan Acute Stroke Trial (SCAST) Study Group: Relation between change in blood pressure in acute stroke and risk of early adverse events and poor outcome. Stroke. 43: 2108–2114. Serrador JM, Sorond FA, Vyas M, Gagnon M, Iloputaife ID and Lipsitz LA (2005). Cerebral pressure-flow relations in hypertensive elderly humans: transfer gain in different frequency domains. J. Appl. Physiol. 98: 151–159. Strandgaard S (1976). Autoregulation of cerebral blood flow in hypertensive patients. The modifying influence of prolonged antihypertensive treatment on the tolerance to acute, drug-induced hypotension. Circulation. 53: 720-727 Tiecks FP, Lam AM, Aaslid R and Newell DW (1995). Comparison of static and dynamic  Cerebral autoregulation measurements. Stroke. 26: 1014–1019. Wang Z, Schuler B, Vogel O, Arras M and Vogel J (2010). What is the optimal anesthetic protocol for measurements of cerebral autoregulation in spontaneously breathing mice? Exp. Brain Res. 207: 249–258. Willie CK, Tzeng YC, Fisher JA and Ainslie PN (2014). Integrative regulation of human brain blood flow. J. Physiol. 592: 841–859. Zhang R, Witkowski S, Fu Q, Claassen JA and Levine BD (2007). Cerebral hemodynamics after short- and long-term reduction in blood pressure in mild and moderate hypertension. Hypertension. 49: 1149–1155. Zlokovic BV (2008). The blood-brain barrier in health and chronic neurodegenerative disorders. Neuron. 57: 178–201.

Billions and Billions :: Economics Economy Reproduction Essays

Billions and Billions Go to Hiroshima of the Frogs It was announced recently that the 6 billionth person was born into the world. The numbers of people born within this past century, are staggering. There have been more people born during this century than any previous one. How much suffering would there be if the Great Depression of the 1930's happened in today's world with so many people vulnerable to famine and starvation? Would the governments be able to provide for all of these people? It is disturbing to note that the earth has such a large population. How many people is too many people? If we look at the animal kingdom, species prey on other species, and populations remain fairly the same unless man has tipped the balance one way or another through industrialization or hunting. Man's presence in the environment seems to have no fail safe device for the overpopulation of people, however. Will we keep breeding and breeding until the capacity of the earth is full? Will there will be enough water and food ? Conditions may become m uch worse than during the the Great Depression. People need to stop having too many children. But how can we deprive someone of their right to reproduce? Natural occurrences such as drought, hurricanes, earthquakes, and volcanoes take a large number of lives. Yet mankind is getting better and better at predicting these events and saving many lives. There are earthquake proof buildings, shelters from hurricanes and Doppler radar to predict floods, tornadoes and hosts of other conditions. Sickness and disease used to be another way Mother Nature kept populations in balance. In today's modern world, we have vaccines, hurricanes and Doppler radar to predict floods, tornadoes and hosts of other conditions. Sickness and disease used to be another way Mother Nature kept populations in balance. In today's modern world, we have vaccines and drugs. People today live nearly twice as long as they did in the beginning of the century. Mankind and its pursuit of progress has surpassed Mother Nature as controller of the planet's destiny. We have an awesome responsibility in today's modern world just to figure out how many people is too many people an d what the capacity of our planet is. Perhaps, subconsciously, our collective brain has determined a way for our populations to remain just below the critical level. We may be taking lessons from the whales.

Essay on Romantics and Merchants in The Merchant of Venice :: Merchant Venice Essays

Romantics and Merchants in The Merchant of Venice  Ã‚   Shakespeare's comedies usually follow a clearly defined pattern. He presents a conflict, and the characters eventually resolve the conflict in a relatively happy ending, which involves marrying off the hero and his entourage to the heroine and her companions, leaving the villain outside the "magic circle" of protagonists. In The Merchant of Venice, Antonio is presented as the hero, and Shylock the villain, but neither is within the circle of marriages at the end of Act V. In fact, Antonio's depression exposed at the beginning of the play seems unresolved at the end, and he goes on his melancholy way, as he supposes he must. Can The Merchant of Venice, then, be considered a true comedy? The strongest argument discounting Merchant as a true comedy is that though Antonio appears to be the major protagonist in the story, he is also as far outside the magic circle as his villain, Shylock. While Bassanio, Portia, and their associated parties marry off at the end of Act V, Antonio is left to his ships and his money, still going about his depressed way. At the beginning of the play, Antonio expresses his dissatisfaction with his situation to his friends. "I hold the world but as the world, Gratiano, a stage where every man must play a part, and mine a sad one" (I.i.81-83). Throughout the play, and Shylock's relentless pursuit of his macabre repayment, Antonio remains in this dreary, defeated state. He seems almost too eager to end his suffering at the hands of his debtors and his apparently lost business. "Grieve not that I am fall'n to this for you," he tells Bassanio in court, "for herein Fortune shows herself more kind than is her custom: it is still her use to let the wretched man outlive his wealth, to view...an age of poverty, from which ling'ring penance of such misery doth she cut me off" (IV.i.278-284). He begs the court to make no more attempts to save his life, comparing such futile endeavors to abate the flood waters or question the wolf's killing of sheep (IV.i.71-84). Completely resigned to his grisly fate, he announces, "I am a tainted wether of the flock, meetest for death. The weakest kind of fruit drops earliest to the ground, and so let me" (IV.i.116-118). Even in Act V, after the dispute with Shylock is decided in Antonio's favor, the melancholy merchant plays no role in the resolution of the play.

Dizziness - The Search for Balance :: Biology Essays Research Papers

On the Search for Balance: A Balancing Act in the Midst of Dizziness Imagine that you are 6 years old again. You are at a playground clutching a pole on the merry-go-round, which is whirling as fast as your friends can run to keep the merry-go-round going. Your friends let go and when you jump off as the merry-go-round stops spinning, you fall because the world seems to wobble out of control. Although you have stopped spinning on the merry-go-round, your brain tells you otherwise. This is the everyday reality for a chronically dizzy person. Following headaches and lower-back pain, dizziness is the third most common medical complaint in the United States. More than 90 million people in the United States will experience a spell of dizziness at some point in their lives (1). Dizziness is a sensation people feel when they lose their sense of spatial orientation. In other words, people feel dizzy when they lose some of their immediate contact with their physical surroundings. A simple disoriented feeling may occur, or one may experience a feeling of movement or of being off balance (2). In most cases, dizziness arises naturally from unusual changes that disrupt the normal feeling of stability. However, a disturbance or a disease in the system that maintains balance can also cause dizziness (3). What could be the cause of this internal lack of balance? Although many forms of dizziness exist, such as Meniere's disease, I am going to focus on what keeps us balanced. The issue of balance first interested me when my mother had gone to see an acupuncturist concerning her high blood pressure. Not only did she come in hopes of finding a cure for her high blood pressure, she also found that she suffers from balance problems. The Chinese guru of an acupuncturist said, "Your whole body is out of balance. Fire blazes within you without equilibrium. You should take a vacation." Apparently, correlation exists between blood pressure and physical balance. Immediately, I became worried and confused. I have been searching for the cause of my mother's problems for some time now. Why is it that she never even noticed that the world was out of balance in her perspective? She always subtly mentioned the feeling of dizziness and the constant coldness of her right shoulder. But I never realized how serious her situation was. I now know that these symptoms are due to an imbalance.

The Beach Descriptive Essay.

Ddirk Mrs. K English 9 A October 24, 2012 Descriptive Essay The look of glee on people faces is the look that you might expect from a child on Christmas day. Why this look? They are on the most amazing place on earth truly it’s a gift from God Himself the â€Å"beach†. The beach is a quiet and peaceful place but at the same time it can be so noisy because the number of people that are on this beach is as if the new iphone 5 is being sold. You just have to allow yourself to become that carefree kid again and let go.As I look out to sea I hear and see the waves crashing down so eager to get to the shore line and I think how can something so beautiful be so deadly at the same time? I am completely unaware of my surroundings as I am lost deep in thought. I hear nothing. Then I am taken aback by the sound of a child’s pure laughter a sound that you would only expect to hear from a child but wait! It’s not a sound of one child laughter it’s a sound of a w hole family completely care free and undaunted by the problem they have, for now the biggest problem they have is who’s sand castle is the best.All they want to do is enjoy these memories in the making and have fun. If you are not careful you might let go of you worries and fear and become a child again. On this sunny beach the hot sand is messed up from people walking on it, making sand castles, hand prints from little kids who are so eager to get themselves messy and my personal favorite foot prints in the sand from big and little kids all alike. I look at the sand as I am walking and I realize just how many sand dollars, seashells and rocks are hidden in the sand just barely visual to a wandering eye.It is like they are playing a game of peek-a-boo with you, not wanting to be caught but also not wanting to be unaware of their surroundings. As I reach out to pick up neat sand dollars that are hairy it’s almost like you are picking up a spider or something that is gro ss, seashells that are so cold that you would think they had been in the freezer, rocks that are so flat and smooth its unreal, and drift wood that is so smooth you don’t get splinters. The sound of seagulls squawking is so loud that you think that you might go crazy if you ere another one squawk again, yet this apart of the beauty of the beach and they too are an amazing part of the beach. Yum†¦ the smell of the beach goes from smelling like a fish hatchery, to the smell of people cooking food on the grill and then you suddenly realize you too are hungry. Its lunch time and it’s time to try to get the sand that is inevitable off of you. I walk into the water unaware that the water is as cold as ice the water reaches my toes first and my instant reaction is to scream.I scream and run out and I am greeted with the stares from the on goers I am instantly embarrassed and suddenly feel like everybody on the beach is staring at me and is wondering what is wrong with th at girl? I should have known that the water would be cold because it’s always cold what was I thinking? I guess I was still in awe of the beauty of the beach I think to myself I will and will never get tired of this amazing and beautiful place for as long as I live. It has been I long day and I am exhausted from such a long day of fun.As the wind on my face is like a fan blowing directly in front of me, cooling of the beach and the people from the heat. I think to myself what makes the beach wonderful is the sound of children’s laughter, the squawking of those annoying seagulls, the smell of the fish hatchery, the grainy feeling of the sand that always seems to get into your mouth no matter what, the smell and the taste of the salty water that you swallow and just the smell of outdoors and wood burning.Those are the things that make up a beach; without those things it would not be near as exciting. As I watch the sunset I realize that the worries of the real world may sometimes not allow a person to become a child again and how sad to think they are on an amazing place with so much beauty which you should be carefree and they can’t because they are not able to let go. I love the beach! I believe it was and is a blessing from God.

Edgware in London to City Road Essay

Using what I have learned from my study materials, I will compare Station Road, Edgware in London to City Road, Cardiff for either their similarities or differences relating to inequalities. Both roads are over 200 years old and have similar layouts. Station Road is located in a neighbourhood and is very similar to City Road as it is extremely busy with high volumes of traffic and also pedestrians shopping and commuting. Along this road there are a few takeaways, cafes, restaurants, newsagents and estate agents, pharmacy, banks, charity shops and also Edgware Underground station. On the one side of the streets is a big shopping centre with more shops inside and well known supermarkets Sainsburyà ¢s and Marks & Spencer. There are similarities on mentioned streets such as road signs, bollards and of course shops and people which make the street what it is. City Road has a large Ethnic minority, on Station road only a small minority are white British, the rest are Asians and East Europ eans. There are also differences between the streets and this essay will use the differences to show how this can create inequalities. Differences are not just there or given, they are made by people in relation to others and by people in relation to material things and their environment. One of the visible inequalities on Station Road is the traffic infrastructure. The numerous controlled crossings help pedestrians access their destinations, but in turn it has an adverse effect for motorists and increases social disorder between motorists and pedestrians as they fight for space among the road. City Road was said to have been a country lane 200 years ago, but by the 1960à ¢s was taken over by car showrooms. Most of them have been replaced by cafes, takeaways and restaurants designed for large numbers of ethnic minority and student population (The Street 2009, scene 1). There are inequalities between local shops and big supermarkets on both streets. On the street I know there are three powerful stores, Sainsburyà ¢s, Marks & Spencer and Lidl. They have large ranges of products, the competitive prices, car parking spaces, they sell more goods for the convenience of people.